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However, the exact mechanism involved in inflicting these adverse effects by NSAIDs is still elusive

In return, dysfunctional eNOS will produce more ROS, and this feedback amplification system forms a vicious cycle, contributing to ECs dysfunction and atherogenesis [153]

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The paper currently receiving a good deal of attention as of this writing (September, 2025), is a Harvard/Mount Sinai study published in 2025