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Description
Lung tissue showed reduced interstitial edema, alveolar congestion, and total damage scores in the I/RB group

Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1
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The discrepancy in temporal stability of FeS clusters as measured by UV-Vis versus NMR analysis was probably due to the different sample holders
