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Conclusion Methaemoglobinaemia is a universal complication of extended NMP with all organs developing severe methaemoglobinaemia in the absence of a prophylactic anti-oxidant

The content of oxidation, glycoxidation, and glycation protein products was measured colorimetrically and fluorimetrically

Nonetheless, there are detectable changes in Tau phosphorylation and expression induced in this TBI model and they are completely unaffected by preinjury supplementation with Immunocal, suggesting that they may represent biochemical changes caused by, or in response to, the primary mechanical injury induced by TBI

10.1016/j.freeradbiomed.2021.12.001 20 ChenW

J., Ursini, F