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doi: 10.1074/jbc.M513231200

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75,420 In particular, whole-exome sequencing (WES) of melanomas with acquired resistance to BRAF inhibitors has revealed that BRAF gene amplification is identified in around 20% of patients, leading to robust upregulation of BRAF protein expression and the reactivation of ERK in response to BRAF inhibition

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however, the accumulated excess intracellular cystine induces disulfide stress, leading to the overproduction of ROS and gradually promoting ferroptosis [16]