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doi: 10.1016/j.pep.2005.03.035

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The cAMP-protein-kinase-A-dependent adenosine inhibition of neutrophil oxidative activity is mediated via the adenosine A 2A receptor [47]

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first reported a polymorphism in GPx1, namely, a substitution at codon 198 of either proline (Pro) or leucine (Leu), Pro198Leu (SNP: 1050450), in lung cancer [87]

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