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Acknowledgements This work was supported by the National Research Foundation of Korea [NRF] grant funded by the Korea government (MSIP) (Grant Number MRC.2008-0062275)

NAC has been extensively used in the treatment of CF to help improve lung function and eliminate mucus due to its mucolytic properties [1], which are attributed to its cysteine residues that break down the sulfhydryl bridges between glycoproteins in mucus, thereby reducing mucus viscosity [28]

While important insights were gained regarding the reduction of glutathione disulfide substrates, the mechanism of non-glutathione disulfide reduction remains highly debated

Additional functional biomarkers such as GPX activity and SelPs levels are also used to evaluate Se-dependent enzymatic functions [2]