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Initial studies show that there are no changes in ductal elongation (5-wk-old mice) or branching morphogenesis (7-wk-old diestrus-staged mice) in Cebpb M as compared to WT mice (Fig 3A and B)

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GLS1 functions as the primary isoform under most conditions, while GLS2 can be induced under specific contexts such as p53 activation, partially compensating for GLS1 activity and thereby adding complexity to its therapeutic targeting [183]

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