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Loss of glial homeostasis, BBB disruption, tanycyte remodelling and neuronal resistance to insulin and leptin lock the system into a state of maladaptive #neuroinflammation

The Pro63, Asp75, and Tyr97 residues are required for GSH oxidation by Og cytAPX1, whereas the corresponding site in At APX1 is composed of Asp63, His75, and His97, and has no GSH binding activity