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glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells The molecular mechanism and therapeutic

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[DOI] [PubMed] [Google Scholar] 23.Murphy AJ, Macdonald LE, Stevens S, Karow M, Dore AT, Pobursky K, et al

glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells The molecular mechanism and therapeutic

doi: 10.1016/0003-2697(90)90208-Q

glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells The molecular mechanism and therapeutic

Spinal injuries Spinal injuries are serious injuries that can have various causes

glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells The molecular mechanism and therapeutic

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glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells The molecular mechanism and therapeutic

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glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells The molecular mechanism and therapeutic

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