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32% HCD collision energy was used for MS2 fragmentation

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Consequently, neuroinflammation in multiple sclerosis originates from a chronic pro-inflammatory state caused by intrinsic metabolic defects in macrophages, while post-stroke inflammation emphasizes the dynamic transition of polarization phenotypes during the acute phase

Imbalanced redox states are increasingly recognized as underlying factors in major diseases, including cancers, neurodegenerative conditions, heart disease, and metabolic disorders [113]