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Post-translational redox control of APR (particularly APR1) marks it out as a significant control point in response to glutathione oxidation (Leustek, 2002)
doi: 10.1104/pp.79.4.1044 48 SuX
For clinicians and people who like to read studies BMC Clin Pharmacol , 2010 (full citation in References below)
As 2 O 3 induces cell death through ROS production [244] by inhibiting the complex I of the respiratory chain (Figure 3) and antioxidant enzymes, such as glutathione peroxidase and thioredoxin reductase [245], as well as activating NADPH oxidase [246]
10.1021/acsnano.0c05499 64 WangL.YuY.WeiD.ZhangL.ZhangX.ZhangG.et al (2021)
