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TCA Cycle Rewiring as Emerging Metabolic Signature of Hepatocellular Carcinoma

For example, lnc19317 exhibited dose-dependent induction in mice but was repressed in rats, whereas lnc45640 showed the opposite trend, being repressed in mice and induced in rats

Because typical early atherosclerotic lesion formation observed in hyperhomocysteinemic animals can be prevented by an antiplatelet therapy with dipyridamole, the authors concluded that platelet-mediated proliferation of smooth muscle cells is a potential cellular mechanism involved in the atherogenicity of hyperhomocysteinemia

In Methods Mol