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Nanomaterials 11 (11), 2816 (2021)

The core argument is that estrogen acts as an endogenous, multi-target inhibitor of ferroptosis, conferring robust anti-ferroptotic capabilities to vascular cells by activating the Nrf2 central antioxidant system, optimizing cellular iron homeostasis (e.g., upregulating FPN1 and inhibiting ferritinophagy), and exerting direct antioxidant effects

Hyperthyroidism In this condition, the thyroid produces excessive hormones

[DOI] [PMC free article] [PubMed] [Google Scholar] 342.Virbasius J

10.1111/jfpp.12456
