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In particular, the renal-type GLS-1 form disrupts GLN-driven oxidative phosphorylation in AML cell lines, thereby preventing tumor growth and inducing apoptosis [55]

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Disruption of its function has been identified in several neurodegenerative diseases, including Alzheimer's, Parkinson's, Huntington's, and prion diseases, as well as in amyotrophic lateral sclerosis (ALS)

Indeed, only baseline Hcy was statistically associated with increased risk of neutropenia and thrombocytopenia on day 8 of treatment cycle 1 (Figure 3)

Factors like stress, poor diet, environmental toxins, and illness can further deplete these levels, creating a need for additional support through supplementation