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Abstract The majority of anticancer agents have a reduced or even complete loss of a therapeutic effect within hypoxic tumors

Glucose-6-phosphate dehydrogenase (G6PD) in the pentose phosphate pathway is the dominant enzyme that produces the required amount of NADPH during the conversion of glucose-6-phosphate to D-glucono-1,5-lactone-6-phosphate [63]

When this phenotype was fully corrected by introducing either HA-ATG2A or HA-ATG2A-3CS, overexpression of ZDHHC11 counteracted the effects of HA-ATG2A but not HA-ATG2A-3CS (Fig

A., Raices, M., Panowski, S

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