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Although current evidence highlights the protective effects of AX on skeletal muscle, several questions remain open

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Assessments of steady-state ATP levels in cells from ME/CFS patients showed conflicting results: decreases were seen in neutrophils, peripheral blood mononuclear cells (PBMCs) and primary skeletal muscle cell cultures [52, 105, 106], while elevated ATP levels were also observed in PBMCs, albeit attributed to nonmitochondrial ATP production [107]

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