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The combination of vancomycin, neomycin sulfate, metronidazole, ampicillin, and other broad-spectrum antibiotics can alleviate choline-induced atherosclerosis in mice, and the mechanism may be related to the inhibition of choline conversion to TMA, the reduction of TMAO levels, and the inhibition of macrophage foaming

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Research demonstrates that MT-1-mediated receptor activation can also engage additional intracellular signaling cascades including MAPK/ERK pathways depending on receptor subtype and cellular context

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