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In contrast, excessive formation of ROS is responsible for disrupted redox homeostasis which in turn leads to oxidative stress and ROS-mediated damage to all important biomolecules including DNA, proteins, and membranes (Liguori et al
It ameliorates intestinal fibrosis in CD by upregulation of p-AMPK expression and suppression of mTOR phosphorylation levels, which enhanced concurrent autophagy and apoptosis in intestinal fibroblasts [22]
N., Singhania R
Ling B, Authier N, Balayssac D, Eschalier A, Coudore F
doi: 10.1042/BJ20030625
