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(60) utilized next generation sequencing (NGS) and computational analysis to demonstrate that miR-23b-3p, acting as a tumor suppressor, can inhibit tumor Gln metabolism by targeting GLS1, thereby enhancing the sensitivity of liver tumor cells to sorafenib
Alternatively, oral administration of NAC led to increased plasma GSH levels as well as effective penetration through the bloodbrain barrier to enhance GSH levels in the brains of animal models [13]
