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Furthermore, cytokines released by astrocytes may bind to their specific receptors in the dopaminergic neurons, such as TNFR1 and 2, and activate proapoptotic mechanisms through the activation of caspase 3, caspase 8, and cytochrome c [10]

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This suggests a potential causal association between Nrf2-regulated impairment of CMA and lung epithelial cell apoptosis mediated by an enhanced UPR, contributing to the development of COPD

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