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Other works have shown that glutathione-ethyl ester is able to effectively prevent oxidative damage of mitochondrial complex I caused by ischemia/reperfusion and thereby reduces the infarct size and improved neurological outcome [34, 35]

In laboratory environments, these peptides are investigated for their interaction with pathways involved in cell migration, connective tissue response and regenerative signalling processes

[DOI] [PubMed] [Google Scholar] 140.Moden O., Mannervik B

[DOI] [PMC free article] [PubMed] [Google Scholar] 172.Yoshikawa H., Sato T., Horikoshi K., Komura M., Nitta N.A., Mitsui A., Koike K., Kodama Y., Takahashi K