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Therefore, at higher levels, ROS are responsible for the damage of most of cellular macromolecules, whereas they exhibit signaling role at moderate levels

demonstrated an Amyloid- 42-induced release of BDNF thereby mediating a neuroprotective effect in cultures of a neuroblastoma cell line (Merlo et al

In females, high estrogen levels provide neuroprotection by reducing oxidative stress, inflammation, and dopaminergic neuron loss [67, 68, 69, 70]

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Conversely, they induce the downstream signaling pathways involving protein kinases of the MAPK and phosphoinositide 3-kinase (PI3K) cascades [12]