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Pattern recognition receptors (PRRs) such as Toll-like receptors (TLRs) expressed on epithelial cells interact with microbiota-derived pathogen-associated-molecular patterns (PAMPs) such as lipopolysaccharide (LPS) to activate myeloid differentiation factor 88 (MYD88)-dependent signaling

However, based on the discussion of the mechanisms of NADPH oxidase activation above, potential mechanisms of deactivation can include the dephosphorylation of phox subunits, activity of GAPs on Rac proteins, and disassembly of the complex

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Some AgNPs with smaller particle sizes can cross the BBB via the paracellular pathway[122]