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doi:10.1016/S0009-8981(00)00264-3 58
10.1016/j.plefa.2018.07.015
Remarkably, treatment with CuII(atsm) significantly mitigated neuronal injury in the ALS mouse model (Liddell et al., 2024)
Severe neonatal hyperbilirubinemia and UGT1A1 promoter polymorphism
Such an hypothesis adds a new function of glutathione and most likely will necessitate a re-evaluation of cell adhesion functions according to their redox-state
