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Together these results indicate that the succination of GSH, but not its detoxification, is responsible for the oxidative stress observed in Fh1-deficient cells GSH succination increases GSH biosynthesis We hypothesized that the chronic depletion of GSH caused by succination could perturb cell metabolism and contribute to a general deregulation of redox homeostasis

The TET enzymes catalyze the 5-mC to 5-hmC, 5-formylcytosine (5fC), and 5-carboxylcytosine (5caC)

Neurodegenerative disease

Effects of 2-year calorie restriction on circulating levels of IGF-1, IGF-binding proteins and cortisol in nonobese men and women: a randomized clinical trial

doi: 10.1016/0954-6111(95)90095-0